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Below we present that DNL343 minimizes ISR activation and neurodegeneration brought on acutely by an optic nerve crush (ONC) damage or chronically by a germline eIF2B LOF mutation in mice. We also clearly show that DNL343 therapy, initiated in a late phase of condition which additional precisely replicates a medical dosing paradigm, reversed these

Name your collection: Title need to be less than characters Pick a set: Struggling to load your selection due to an mistakeThree differing types of animal designs are currently recognized to the exploration of pathologic designs of MS: Viral models like Theiler's murine encephalomyelitis virus (TMEV), products induced by toxic agents like cuprizone